Maria tokuyama

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Mission Statement: As citizens of the scientific biosphere, we value representation of diverse individuals who advance science and enrich the scientific community. We welcome scientists from underrepresented backgrounds, and as a team, we are committed to reforming institutional culture and policies that perpetuate racism and exclusion in science. Lab Research: The core of our research is to understand how chronic interaction between viruses and the immune system impacts immunity at steady state and during inflammation. We study endogenous retroviruses ERVs , which are viral sequences in our genome that originated from exogenous viruses and have undergone an evolutionary arms race with the host for millions of years. We currently have multiple projects aimed at uncovering how the interaction between ERVs and the immune system impacts antiviral immunity and excessive inflammation in autoimmunity. We rely on multidisciplinary approaches and novel tools in virology, immunology, and computational genomics.

Maria tokuyama

Google Scholar. Tokuyama lab. Tokuyama has always been interested in the interaction between viruses and the immune system. As an undergrad at University of California, Irvine, she worked in an infectious disease lab that focused on HIV research and obtained her B. Tokuyama obtained her Ph. Tokuyama trained at Yale University in the Department of Immunobiology, where she became fascinated by endogenous retroviruses and established the foundation for the research that she will carry out at UBC. In the past year, Dr. The overarching goal of our lab is to identify immunological mechanisms that underlie heterogeneity in disease outcomes. Our lab seeks to understand how interactions between the immune system and the virome influence outcomes of viral infection and autoimmunity. In particular, we are focused on uncovering immunomodulatory functions of endogenous retroviruses ERVs , which are retroviral sequences that make up a large fraction of the human genome. Using clinical samples, in vivo mouse models and 2D and 3D cell cultures, we seek to uncover how endogenous viral proteins affect immune cell signaling and function in health and disease through transcriptomic, proteomic and functional assays. Furthermore, we will employ computational analysis methods to study endogenous viral elements to deepen our understanding of how these elements are regulated and their potential roles in clinical outcomes.

Furthermore, we will employ computational analysis methods to study endogenous viral elements to deepen our understanding of maria tokuyama these elements are regulated and their potential roles in clinical outcomes. Support us.

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Maria tokuyama

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We study endogenous retroviruses ERVs , which are viral sequences in our genome that originated from exogenous viruses and have undergone an evolutionary arms race with the host for millions of years. Citations per year. Research Faculty. MedRxiv, About MBIM. Assistant Professor. View all. Life Sciences Institute. We rely on multidisciplinary approaches and novel tools in virology, immunology, and computational genomics. PloS one 3 10 , e , Privacy Terms Help. Furthermore, we will employ computational analysis methods to study endogenous viral elements to deepen our understanding of how these elements are regulated and their potential roles in clinical outcomes. Student Resources. We rely on multidisciplinary approaches and novel tools in virology, immunology, and computational genomics.

The lab hopes to contribute towards development of immune modulators to treat infections and chronic inflammatory diseases.

MedRxiv , Add co-authors Co-authors. Ultimately, our goal is to identify novel endogenous viral factors that underlie immunity and contribute towards development of immune modulators to treat infections and chronic inflammatory diseases. Lab Research: The core of our research is to understand how chronic interaction between viruses and the immune system impacts immunity at steady state and during inflammation. Ultimately, our goal is to identify novel endogenous viral factors that underlie immunity and contribute towards development of immune modulators to treat infections and chronic inflammatory diseases. Lab Tel. This "Cited by" count includes citations to the following articles in Scholar. Citations per year. Email address for updates. Infection and immunity 73 8 , , Office Tel.